View source for Exudative interstitial inflammations

[[Inflammation]] is a phylogenetically established response of the organism to the action of a harmful substance. It has a defensive and reparative function. It manifests locally and overall.

== Composition ==
It has parts:
*alterative
*exudative
*proliferative
*immune
                    
Exudation is the formation of an inflammatory effusion, exudate. Expansion of capillaries, increased permeability for [[Plasmatic proteins|proteins (albumins, globulins, fibrinogen)]] , [[erythrocytes]] and cells of the inflammatory infiltrate ( [[Neutrophilic granulocytes|neutrophils]] , [[macrophages]] , [[Lymphocyte|lymphocytes]] , [[plasma cells]] , [[capillary endothelial cells]] , [[fibroblasts]] ). According to the predominant component, the exudate is: serous, fibrinous, purulent, serofibrinous, hemorrhagic...

== Serous interstitial inflammation ==
Serous exudate – clear, straw-yellow, thin fluid.

Example: [[Urticaria|hives]] , [[Allergy|allergic reactions]] (tongue, lip after wasp, bee sting).

== Suppurative interstitial inflammation ==
Purulent exudate - viscous, thick liquid, whitish-yellow, yellow-green, blue, red. It destroys tissues - [[Necrosis|collicative necrosis]] !

=== Abscess ===
[[Abscess]] is a cavity filled with pus, a circumscribed form.

acute
chronic – bounded by a pyogenic (pus-forming) membrane
Healing: the [[fistula]] collapses , the cavity either collapses and scars or does not collapse (e.g. in bone), the pyogenic membrane will still produce pus and the fistula becomes chronic (risk of secondary [[amyloidosis]] ).

Cause: [[Staphylococcus]] .

=== Phlegmon ===
Phlegmon is an acute unbounded form of inflammation that spreads in the skin, subcutaneous tissue and other soft tissues. The tissue is sludgy and oozing droplets of pus. It manifests as a widespread, warm, red to red-purple spreading edema of the skin and subcutaneous tissue, which is not as sharply demarcated as in [[erysipelas]] and is much more painful.<ref>{{Cite
| type = article
| surname1 = MUDR. Čapková
| name1 = Štěpánka
| article = Hnisavé kožní infekce (pyodermie)
| journal = POSTGRADUÁLNÍ MEDICÍNA
| year = 2009
| number = 10
| volume = 6
| pages = 38-43
| issn = 1212-4184
}}</ref> It often arises as a complication of small wounds and abrasions in children. It is also among the postoperative complications of wound healing.

Course: the tendency to [[Wound healing|healing]] is minimal, rather it spreads.

Cause: Staphylococcus, [[Streptococcus]] <ref>{{Cite
| type = article
| surname1 = MUDR. Čapková
| name1 = Štěpánka
| article = Hnisavé kožní infekce (pyodermie)
| journal = POSTGRADUÁLNÍ MEDICÍNA
| year = 2009
| number = 10
| volume = 6
| pages = 38-43
| issn = 1212-4184
}}</ref>.

== Fibrinous interstitial inflammation ==
The bearings are microscopic, they are not visible.

Examples: rheumatic fever , polyarteriitis nodosa , lupus .

Healing: minor scars.

== Gangrenous interstitial inflammation ==
Rotting, gray-green, foul-smelling, decaying tissue.

Examples: appendicitis , tumors , aspiration pneumonia (inhaled rotten tooth, piece of tumor).

Course: the appendix bursts and the patient dies of stercoral peritonitis , he also dies of lung gangrene .
<noinclude>
== Links ==
=== Related articles ===
* [[Inflammation]]
* [[Macroscopic manifestations of inflammation]]
* [[Microscopic manifestations of inflammation]]
=== References ===
* {{Cite
| type = book
| surname1 = Stříteský
| name1 = Jan
| title = Patologie
| publisher = Epava
| edition = 1
| year = 2001
| isbn = 80-86297-06-3
}}
=== References ===
<references />
</noinclude>

[[Category:Pathology]]