Circulatory shock: Difference between revisions
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** VÍZEK, Martin. ''Repetitorium'' [online]. [cit. 2011-11-02]. <https://web.archive.org/web/20130512032641/http://pf.lf2.cuni.cz/vyuka/repetitorium.html>. | ** VÍZEK, Martin. ''Repetitorium'' [online]. [cit. 2011-11-02]. <https://web.archive.org/web/20130512032641/http://pf.lf2.cuni.cz/vyuka/repetitorium.html>. | ||
[[ | [[Category:Pathophysiology]] | ||
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Revision as of 18:46, 16 March 2023
This article has been translated from WikiSkripta; ready for the editor's review.
Inability to maintain adequate blood flow through peripheral tissues and arterial blood pressure.
Cause:
- The imbalance between the volume of blood and the volume of the vascular bed caused by:
- reduced blood volume (hypovolemic shock, cold shock)
- by increasing the capacity of the vascular bed (distributive shock, warm shock)
- Hypovolemic shock:
- Hemorrhage
- Burns (hematocrit, toxins, pain)
- Dehydration (AB balance)
- Distribution shock:
- Anaphylaxis (bronchoconstriction)
- Sepsis (fever, toxins)
- Neurogenic vasodilation
The shock phase:
- initial (activation of compensatory mechanisms)
- Progressive (failure of compensatory mechanisms)
Consequences:
- Tissue ischemia and microcirculation
- Consequences of damage to individual organs
Treatment:
- Maintaining the volume of circulating fluid
- Sympathomimetics
- Corticosteroids
Links:
Related articles:
External links:
Source:
- VÍZEK, Martin. Repetitorium [online]. [cit. 2011-11-02]. <https://web.archive.org/web/20130512032641/http://pf.lf2.cuni.cz/vyuka/repetitorium.html>.
