Facial hemispasm

With facial hemispasm there are involuntary unilateral, painless, spastic contractions of mimic muscles.

• typically start at the m. orbicularis oculi and descend to the mimic muscles of the face and periorally;
• atypical form: starts buccally and spreads upward;
• most often caused by compression of the REZ (root entry zone) facial nerve a. cerebelli inferior anterior.

Differential diagnosis

• facial myokymia (continuous spasm, may be a manifestation of a tumor brain stem / multiple sclerosis);
• blepharospasm;
• hemifacial spasm persists in sleep.

Treatment of facial hemispasm

• microvascular decompression n. VII (success rate 70-90%) - the main risk of surgery hearing damage (manipulation of n . VIII, necessary intraoperative monitoring BAEP), other risks are injury to the decompressed n. VII or damage to the cerebellum;
• "elderly and high-risk patients": local application of botulinum toxin to the muscle affected by the spasm, the effect lasts for about 5 months, after which the application must be repeated.

Links

Related Articles

• Facial nerve palsy
• Trigeminal neuralgia

References

• {{#switch: book

|book =

  Incomplete publication citation. SAMESH, M, et al. Neurosurgery. Prague : Jessenius Maxdorf, 2005. 978-80-7262-438-6.

|collection =

  Incomplete citation of contribution in proceedings. SAMESH, M, et al. Neurosurgery. Prague : Jessenius Maxdorf, 2005. {{
  #if: 80-7345-072-0 |978-80-7262-438-6} }
  |article = 
  Incomplete article citation.  SAMESH, M, et al. 2005, year 2005, 

|web =

  Incomplete site citation. SAMESH, M, et al. Jessenius Maxdorf, ©2005. 

|cd =

  Incomplete carrier citation. SAMESH, M, et al. Jessenius Maxdorf, ©2005. 

|db =

  Incomplete database citation. Jessenius Maxdorf, ©2005. 

|corporate_literature =

  SAMESH, M, et al. Neurosurgery. Prague : Jessenius Maxdorf, 2005. 978-80-7262-438-6} }