HelpClose
If you encounter any technical issues as you edit, please report them.
3 noticesClose

Warning: You are not logged in! We appreciate you want to improve our articles. It is possible to save your changes anonymously to WikiLectures, however, your IP address will be recorded in this page's edit history. But if you want to log in or create a new account to save your contribution under your user name, you can do so.

You do not have permission to edit this page, for the following reason:

You must confirm your email address before editing pages. Please set and validate your email address through your user preferences.

You are using a browser which is not officially supported by this editor.

Bradycardia

From WikiLectures

Bradycardia is a slowing of the heart rate below the physiological limit, most commonly considered to be bradycardia at less than 60 beats/min (in adults). Bradycardia can occur in sleep or in a state of physical and mental relaxation. It is often present physiologically under resting conditions in trained athletes. The opposite of bradycardia is tachycardia.

Bradycardia:


Bradycardia 2:



Pathological causes of bradycardia include hypothyroidism, disease of the sick sinus syndrome (SSS), disengagement of the sinus node, fluctuations of the ions (hyperkalaemia), intracranial injury, myocardial infarct, inflammation of the heart.

Bradycardia therapy (for men <60 beats, for women <65 beats):

  1. atropine i.v.
    • operation:
      • the greatest effect on the sinus and atrioventricular nodes, the distal efficiency decreases
      • with complete AV blockages with broad QRS effectiveness little/none
    • mechanism: Vagolytic
    • effect: short-term about 30 min, cannot be given long-term
    • indication: in bradycardia <60/min.
    • dose: 0.5 mg i.v., repeat max to 3 mg i.v., but not as a bolus
  2. izoprenaline in i.v. infusion
    • operation: catecholamins
    • mechanism: sympathomimetic agent
    • dose: Isoprenaline 5 ųg/min
    • effect: at the sinusoidal node, atrioventricular node, even at the more distant centers
  3. discontinue bradyarrhythmic drugs: beta blockers or antiarrhythmics
  4. therapies for possible thyroid hypofunction
  5. (temporary) cardiostimulation

12 lead sinus bradycardia.JPG



References[edit | edit source]

Referenced articles[edit | edit source]

External references[edit | edit source]

Sources[edit | edit source]

non-existent now
  • ŠEVČÍK, Pavel – MATĚJOVIČ, Martin. Intenzivní medicína.. 3., přeprac. a rozš. edition. Praha : Galén, spol. s r.o., 2014. 1195 pp. ISBN 978-80-7492-066-0.


Bradycardia is a slowing of the heart rate below the physiological limit, most commonly considered to be bradycardia at less than 60 beats/min (in adults). Bradycardia can occur in sleep or in a state of physical and mental relaxation. It is often present physiologically under resting conditions in trained athletes. The opposite of bradycardia is tachycardia.

Bradycardia:

START_WIDGET"'-5c1e00eae87c6f1aEND_WIDGET

Bradycardia 2:

START_WIDGET"'-41d1bac1e1555c73END_WIDGET


Pathological causes of bradycardia include hypothyroidism, disease of the sick sinus syndrome (SSS), disengagement of the sinus node, fluctuations of the ions (hyperkalaemia), intracranial injury, myocardial infarct, inflammation of the heart.

Bradycardia therapy (for men <60 beats, for women <65 beats):

  1. atropine i.v.

    • operation:

      • the greatest effect on the sinus and atrioventricular nodes, the distal efficiency decreases

      • with complete AV blockages with broad QRS effectiveness little/none

    • mechanism: Vagolytic

    • effect: short-term about 30 min, cannot be given long-term

    • indication: in bradycardia <60/min.

    • dose: 0.5 mg i.v., repeat max to 3 mg i.v., but not as a bolus

  2. izoprenaline in i.v. infusion

    • operation: catecholamins

    • mechanism: sympathomimetic agent

    • dose: Isoprenaline 5 ųg/min

    • effect: at the sinusoidal node, atrioventricular node, even at the more distant centers

  3. discontinue bradyarrhythmic drugs: beta blockers or antiarrhythmics

  4. therapies for possible thyroid hypofunction

  5. (temporary) cardiostimulation


References

Referenced articles

External references

Sources

non-existent now
  • ŠEVČÍK, Pavel – MATĚJOVIČ, Martin. Intenzivní medicína.. 3., přeprac. a rozš. edition. Praha : Galén, spol. s r.o., 2014. 1195 pp. ISBN 978-80-7492-066-0.