Retroperitoneal fibrosis: Difference between revisions
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[[File:Ultrasonography of hydronephrosis due to ureteropelvic junction obstruction.jpg|thumb|Sonographic image of dilatation of the calicopelvic system of the kidney]] | [[File:Ultrasonography of hydronephrosis due to ureteropelvic junction obstruction.jpg|thumb|Sonographic image of dilatation of the calicopelvic system of the kidney]] | ||
'''Retroperitoneal fibrosis''' is '''an inflammatory proliferation''' [[Of the connective tissue of the retriperitoneum|of the connective tissue of the retroperitoneum]] , which by its action affects or oppresses the surrounding organs, most often [[Ureter|the ureters .]] | '''Retroperitoneal fibrosis''' is '''an inflammatory proliferation''' [[Of the connective tissue of the retriperitoneum|of the connective tissue of the retroperitoneum]], which by its action affects or oppresses the surrounding organs, most often [[Ureter|the ureters.]] | ||
== Etiology == | == Etiology == | ||
We distinguish between primary and secondary fibrosis. The etiology '''of primary''' fibrosis (also Ormond disease) is unclear, an autoimmune reaction is assumed. In contrast, the causes '''of secondary''' fibrosis are many, for example drugs (methyldopa, phenacetin, beta blockers, LSD), tumors, infections and others. | We distinguish between primary and secondary fibrosis. The etiology '''of primary''' fibrosis (also Ormond disease) is unclear, an autoimmune reaction is assumed. In contrast, the causes '''of secondary''' fibrosis are many, for example drugs (methyldopa, phenacetin, beta blockers, LSD), tumors, infections and others. | ||
The disease occurs between the ages of 40 and 60 with a preference for men (2.9:1) | The disease occurs between the ages of 40 and 60 with a preference for men (2.9:1).<ref>{{Cite | ||
| type = article | |||
| surname1 = Fedorko | |||
| name1 = Michal | |||
| surname2 = Krhovský | |||
| name2 = Miroslav | |||
| article = Perirenální retroperitoneální fibróza | |||
| journal = Urologie pro praxi | |||
| url = [https://www.urologiepropraxi.cz/pdfs/uro/2009/05/11.pdf|zde] | |||
| year = 2009 | |||
| volume = 10 | |||
| the_year = 5 | |||
| pages = 299-301 | |||
| issn = 1213-1768 | |||
}}</ref> | |||
== Manifestations and diagnosis == | == Manifestations and diagnosis == | ||
The most common manifestation is external '''compression of the ureter''' with a decline in renal function to the point of [[renal failure]] . Branches of large vessels or nerves can also be obturated. At the same time, we can observe non-specific symptoms such as nausea, weight loss or subfebrile. | The most common manifestation is external '''compression of the ureter''' with a decline in renal function to the point of [[renal failure]]. Branches of large vessels or nerves can also be obturated. At the same time, we can observe non-specific symptoms such as nausea, weight loss or subfebrile. | ||
In diagnosis, we use '''imaging methods''' (USG, CT and [[excretory urography]] ) to evaluate the condition of the urinary tract, [[Calicopelvic system|dilatation of the calicopelvic system]] and the general condition of the retroperitoneum. We examine renal function and autoimmune antibodies. In case of uncertainty, a biopsy will confirm the diagnosis. | In diagnosis, we use '''imaging methods''' (USG, CT and [[excretory urography]]) to evaluate the condition of the urinary tract, [[Calicopelvic system|dilatation of the calicopelvic system]] and the general condition of the retroperitoneum. We examine renal function and autoimmune antibodies. In case of uncertainty, a biopsy will confirm the diagnosis. | ||
== Treatment == | == Treatment == | ||
The basis of the treatment is the restoration of urine outflow from the kidney, which leads to an improvement in its function. We use [[Derivace moče – dočasná, trvalá|ureteral stents or nephrostomy]] for ''derivation'' . The causal treatment of the primary form is [[Immunosupresants|immunosuppressants]] . Another option is surgical release of the ureter. | The basis of the treatment is the restoration of urine outflow from the kidney, which leads to an improvement in its function. We use [[Derivace moče – dočasná, trvalá|ureteral stents or nephrostomy]] for ''derivation''. The causal treatment of the primary form is [[Immunosupresants|immunosuppressants]]. Another option is surgical release of the ureter. | ||
== Links == | == Links == | ||
| Line 21: | Line 35: | ||
* [[Diagnostic imaging methods in retroperitoneum examination]] | * [[Diagnostic imaging methods in retroperitoneum examination]] | ||
=== | === Literature === | ||
* | *{{Cite | ||
| type = article | |||
| surname1 = Sobotka | |||
| name1 = Roman | |||
| surname2 = Hanuš | |||
| name2 = Tomáš | |||
| article = Primární retroperitoneální fibróza | |||
| journal = Urologie pro praxi | |||
| url = https://www.urologiepropraxi.cz/pdfs/uro/2006/04/02.pdf | |||
| year = 2006 | |||
| volume = 4 | |||
| pages = 156-159 | |||
| issn = 1213-1768 | |||
}} | |||
*{{Cite | |||
| type = article | |||
| surname1 = Kašík | |||
| name1 = Josef | |||
| others = yes | |||
| article = Primární retroperitoneální fibróza - Morbus Ormond | |||
| journal = Urologie pro praxi | |||
| url = https://www.urologiepropraxi.cz/pdfs/uro/2008/03/06.pdf | |||
| year = 2008 | |||
| the_year = 9 | |||
| volume = 3 | |||
| pages = 128-132 | |||
| issn = 1213-1768 | |||
}} | |||
=== Reference === | |||
<references /> | |||
</noinclude> | |||
[[Category:Urology]] | |||
[[Category:Missing link to the original WikiScripta article]] | |||
Latest revision as of 16:16, 5 June 2023
Retroperitoneal fibrosis is an inflammatory proliferation of the connective tissue of the retroperitoneum, which by its action affects or oppresses the surrounding organs, most often the ureters.
Etiology[edit | edit source]
We distinguish between primary and secondary fibrosis. The etiology of primary fibrosis (also Ormond disease) is unclear, an autoimmune reaction is assumed. In contrast, the causes of secondary fibrosis are many, for example drugs (methyldopa, phenacetin, beta blockers, LSD), tumors, infections and others.
The disease occurs between the ages of 40 and 60 with a preference for men (2.9:1).[1]
Manifestations and diagnosis[edit | edit source]
The most common manifestation is external compression of the ureter with a decline in renal function to the point of renal failure. Branches of large vessels or nerves can also be obturated. At the same time, we can observe non-specific symptoms such as nausea, weight loss or subfebrile.
In diagnosis, we use imaging methods (USG, CT and excretory urography) to evaluate the condition of the urinary tract, dilatation of the calicopelvic system and the general condition of the retroperitoneum. We examine renal function and autoimmune antibodies. In case of uncertainty, a biopsy will confirm the diagnosis.
Treatment[edit | edit source]
The basis of the treatment is the restoration of urine outflow from the kidney, which leads to an improvement in its function. We use ureteral stents or nephrostomy for derivation. The causal treatment of the primary form is immunosuppressants. Another option is surgical release of the ureter.
Links[edit | edit source]
Related Articles[edit | edit source]
Literature[edit | edit source]
- SOBOTKA, Roman – HANUŠ, Tomáš. Primární retroperitoneální fibróza. Urologie pro praxi [online]. 2006, vol. 4, p. 156-159, Available from <https://www.urologiepropraxi.cz/pdfs/uro/2006/04/02.pdf>. ISSN 1213-1768.
- KAŠÍK, Josef. , et al. Primární retroperitoneální fibróza - Morbus Ormond. Urologie pro praxi [online]. 2008, y. 9, vol. 3, p. 128-132, Available from <https://www.urologiepropraxi.cz/pdfs/uro/2008/03/06.pdf>. ISSN 1213-1768.
Reference[edit | edit source]
- ↑ FEDORKO, Michal – KRHOVSKÝ, Miroslav. Perirenální retroperitoneální fibróza. Urologie pro praxi [online]. 2009, y. 5, vol. 10, p. 299-301, Available from <[https://www.urologiepropraxi.cz/pdfs/uro/2009/05/11.pdf>. ISSN 1213-1768.
