Adenoid vegetations
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Adenoid vegetation

The term adenoid vegetation (AV) refers to the pathological enlargement of the nasopharyngeal tonsil (tonsilla pharyngea), which occurs most often as a result of chronic rhinosinusitis. It is a childhood disease affecting both sexes.

The nasopharyngeal tonsil is also referred to as the so-called third tonsil (next to the palatine tonsils). It is part of Waldeyer's lymphoepithelial circuit. Its function is to participate in the body's defense against infection . It is most developed in children aged 4–7 years, after which it gradually involves.

  • If it is pure hyperplasia , the tonsil is uniformly enlarged and there is no secretion in the nasopharynx.
  • As for the enlargement caused by infection , the enlargement is irregular, the surface of the tonsil is furrowed and covered with secretion, which is also present in the nasopharynx and the nose.

Meaning and development of AV

  • Lymphatic tissue rich in secretory glands;
  • Meaning: participation in the formation of immunity, production of immunoglobulins and lymphocytes, part of the lymphoepithelial barrier;
  • Hyperplasia corresponds to the immunobiological activity of childhood (natural hypertrophy from the activity of lymph tissue and the secretory apparatus);
  • Hyperplasia is stimulated by stress on the immunological system, poor nutrition, repeated inflammation of the upper respiratory tract, hormonal influences (inflammatory hypertrophy with proliferation of fibrous stroma);
  • Greatest between 3 and 5 years of age;
  • From the age of 7 it involutes and disappears, but it can persist;
  • For the formation of overall immunity, the importance of the nasal tonsil is marginal and it can be replaced by other organs of the Waldeyer circuit;
  • The most common infectious focus in children in the ENT area.[1]

Clinical signs

  • nasal obstruction - children breathe through their mouths (especially during sleep), night snoring, rhinolalia clausa;
  • sleep apnea syndrome
  • facies adenoidea – characteristic facial expression (open mouth, indistinct nasolabial fold, upper lip retraction, exposed upper incisors, flattened cheeks, arched and narrow hard palate – Gothic palate);
  • recurrent diseases from colds , rhinosinusitis ;
  • recurrent mesotitis ;
  • hearing loss – an enlarged tonsil covers the mouth of the Eustachian tube, a negative pressure will be created in the middle ear, the eardrum will be pushed in and there will be catarrhal inflammation with exudation;
  • may be bedwetting
  • mechanical dysphagia and failure to thrive.

Diagnostics

Differential diagnosis

Benign tumors and cysts of the epipharynx, especially juvenile angiofibroma and antrochoanal polyp, must be differentiated in terms of differential diagnosis .

Treatment

  • adenotomy - curettage of the nasopharynx with a curette under general inhalation or intubation general anesthesia; endoscopic adenotomy;
  • remediation of HCD inflammations ;
  • nose breathing rehabilitation .


Links

Related Articles

Source

References

  • KLOZAR, Jan, et al. Speciální otorinolaryngologie. 1. edition. Prague : Galén, 2005. 224 pp. ISBN 80-7262-346-X.
  • HAHN, Aleš, et al. Otorinolaryngologie a foniatrie v současné praxi. 1. edition. Prague : Grada, 2007. 390 pp. ISBN 978-80-247-0529-3.
  1. KOLÍN, Jan. Adenoidní vegetace. Vox Pediatriae [online]2003, y. 3, vol. 2, p. 28, Available from <http://www.detskylekar.cz/cps/rde/xbcr/dlekar/2003_vox2.pdf>.