Vascular dementia
From WikiLectures
- brain damage by heart attacks
- at least 100ml of tissue is damaged to cause dementia
- with larger CMPs and smaller volume
- mainly small arteries, thrombosis, embolism and bleeding
- often arise suddenly and quickly, often with a history of stroke or ischemic heart disease
- typical – fluctuating course in a short time horizon
- the personality is preserved for a long time → they are aware of the disease for a long time → frequent depression
- often additional neurological symptoms
- frequent polymobility, sometimes even delirium
- 15-30% of dementia
Vascular dementia with acute onset[edit | edit source]
- after one more extensive heart attack, mainly in the areas of cognitive functions
- thalamus, gyrus angularis, frontal area
Multi-infarct dementia[edit | edit source]
- multiple small infarcts, mainly in the cortical area
- often together with hypertension, smokers, hyperlipidemia
Subcortical vascular dementia (Binswanger's disease)[edit | edit source]
- on the basis of hypertensive encephalopathy
- involvement of white matter infarcts - demyelination
- the course is different
Other types of ischemic-vascular dementias[edit | edit source]
- sometimes status lacunaris is isolated separately – relatively mild dementia on the basis of hypertension
Diagnostics[edit | edit source]
- the course and pre-disease, as well as the image, are important
- risk factors - hypertension, stroke, smoking...
Therapy[edit | edit source]
- complex as in other dementias
- rehabilitation of intercurrent diseases - mainly hypertension, diabetes mellitus,...
- antioxidants, nootropics…
- additionally anticoagulants – ASA, heparin
Links[edit | edit source]
References[edit | edit source]
BENEŠ, Jiří. Studijní materiály [online]. [cit. 10.03.2010]. <http://jirben.wz.cz>.