Examination of the child's skin and skin adnexa
Skin[edit | edit source]
(lat. cutis, greek. derma)
It is one of the smaller ones organs human bodies. It has mass approximately 7% body weight.
Between her function include:
- Function protective ( barrier ) of her acidic pH 4.5-6 slows down multiplication microorganisms (mainly yeast and mold). In addition and mechanic prevents injury and penetration microorganisms into the body.
- Thermoregulatory : Uses evaporation (sweating), radiation and convection (conduction) to relieve body excess heat. In the cold environment is running out to slowdown these physical events (reduction circulation and prevention losses heat).
- Immune : Immune function in the skin fulfills especially Langerhans's cells which they have ability phagocytosis and presentation of antigen.
- Excretory-resorptive : To function skin belongs and production sweat and sebum. Possible ones resorptive properties are used especially in local application medicines.
- Perceptual : Located in the skin and nervous endings and misc second of receptors.[1]
Investigation skin[edit | edit source]
Anamnesis[edit | edit source]
Contains the same requisites like any one other anamnesis (NO, RA, PSA, OA, FA, Abuse, GA, …), in addition we put regard to skin speeches disease. We ask about course , changes in time and intensity. We are trying to find evocative the cause, possibly periodicity.
Physical examination[edit | edit source]
- Color skin
Deviations | Causes |
---|---|
decrease or loss of pigment | albinism , vitiligo, conditions after inflammations skin (hypo/ hyperpigmentation ) |
cyanosis | presence more than 50 g/l of reduced hemoglobin u |
icterus | rise serum levels [[bilirubin] ]u |
nevi | " Mongolian spot " gray-blue , naevus flammeus , hemangioma y, pigmented nevus |
stains colors white coffee (café-au- lait ) | neurofibromatosis ,McCune-Albright syndrome |
haematomas , petechiae | disorders hemocoagulation , thrombocytopenia , vasculitis |
- Cutaneous efflorescence primary
Type efflorescence | Characteristics |
---|---|
macula | petty bearing (below 1 cm) in level skin , in whose range is leather colorfully changed to Spots they are efflorescence exceeding 1 cm |
papule | petty palpable bearing (below 1 cm ) projecting above level skin surface Nodules are formations over 1 cm |
plaque | elevated flat lesion ( over 1 cm), whose the surface is larger than elevation , arises direct or merging papule |
vesicle | small formation (below 1 cm) formed cavity with contents fluid Bula is a formation the same character , but exceeding 1 cm |
pustule | unit similar vesicles , but with purulent content |
cyst | bounded thin-walled unit localized deeper in the skin with liquid or semi-liquid content and covered normal epidermis |
- Cutaneous efflorescence secondary
- excoriation ,
- ulceration ,
- peeling,
- crusts,
- fissures ,
- scars. [1]
Hair and nails (skin adnexa)[edit | edit source]
State the skin under the armpit is also important diagnostic by the flag which can at not thorough examination remain hidden. It often does manifests psoriasis or systemic lupus. By the hair we evaluate speed growth, brittleness, fraying ends and alopecia. These pathology more often occur in autoimmune diseases. On the nails you are we notice nail disc, surrounding skin and condition himself nail _ e.g. _ dimples in psoriasis, changes thickness at various fungal affections.[1]
Maternity signs of newborns[edit | edit source]
Maternal signs newborns you can divided into 3 groups :
- pigmented (congenital melanocytic Nevus, Mongolian stain);
- vascular (hemangioma, naevus flammeus, naevus simplex);
- incurred on basis abnormal development.
Some maternal signs require attention for possible malignant potential, with some it is necessary investigate further possible associated defects.
Congenital melanocytic nevus[edit | edit source]
Congenital melanocytic nevus (naevus giganteus, nevus swimsuit) probably arise as a result disturbed migration precursors melanocytes in neural bar. Yours essentially they are these nevi hamartomas. They occur in up to 2% of newborns, in exceptional cases and in form the so- called huge ones congenital melanocytic nevus. They tend to brown whose black color, they can be flat or prominent and frequent they are present terminal hair. They are potentially malignant - the risk of malignancy increases with size and number nevus. Dermatological monitoring is suitable for pigmented ones nevus bigger ones than 0.5 cm. With growth of the child they can nevi to change its appearance - at change colors whose shape is suitable examination for exclusion melanoma. Prophylactic removal lesion does not guarantee protection before development melanoma, therefore it is dermatological tracking appropriate and after removal congenital melanocytic nevus. Congenital nevi they can be associated with a number of others disorders, such as _ _ neurofibromatosis or leptomeningeal proliferation melanocytes leading to hydrocephalus and others CNS defects (neurocutaneous melanosis ).[2][3]
Mongolian stain[edit | edit source]
Mongolian spot ( dermal melanosis ) is blue-grey hyperpigmentation most often in the lower ones parties stern whose on buttocks, which you can easily confused with hematoma. It is benign affection. It occurs more often in children Asian and Hispanic origin and black people. It has smooth surface and comparatively clearly boundary. Histologically can be in the pars reticularis coria demonstrate fusiform melanocytes oriented parallel to the skin by the surface, the epidermis is normal, they can be present and melanophages. With growing by age fading occurs, namely most often under 2 years of age. Therapy it isn't necessary. To the malignant changes does not occur.[3][2]
Hemangioma[edit | edit source]
Hemangiomas can develop whenever during the first ones months of life. They are present in 10% of children in one a year age. Children's hemangiomas they have tendency to disappear - 50% will disappear up to 5 years of age, 90% up to 10 years of age. In place hemangioma can then arise atrophy, telangiectasia, hypopigmentation whose scar. Multiple cutaneous hemangiomas they can be accompanied hemangiomas liver and digestive tract with risk obstruction whose bleeding. Rare they lead extensive and numerous hemangiomas to cardiac failure on basis a big one cardiac issues.[2]
Naevus flaming[edit | edit source]
Nevus flammeus (port-wine stain) is flat dark red until purple vascular lesion present already at birth. It is relatively rare - occurs in 0.3 % of newborns, his the basis is dilation vessel in corium. Unlike hematomas usually it does not disappear, on the contrary can still darken. It doesn't require treatment. In the first a year of life is possible therapy laser to lighten lesions. At occurrence in area 1. branches trigeminal nerve – nerve ophthalmicus (V1) is suitable ocular examination for exclusion glaucoma , which can occur independently or within Sturge-Weber syndrome ( defined triad glaucoma , epilepsy , naevus flammeus and tends to associated with the present angiomas brain and meninges ).[2]
Naevus simple[edit | edit source]
Naevus simplex (stork bites, angel's kisses, salmon patches) is salmon- like colored lesion caused by telangiectasias. It occurs in 33% of newborns. He lives in the area eye, hair parts head, neck and compression fades. It occurs frequently bilaterally or symmetrically, on difference from naevus flammeus. It is benign lesions. 40% of these nevus disappears during newborn period, most disappear within 18 months of age.[2]
Links[edit | edit source]
Related articles[edit | edit source]
External links[edit | edit source]
Reference[edit | edit source]
- ↑ a b c LEBL, Jan – HEJCMANOVÁ, Ludmila. Preclinical Pediatrics. 1. edition. Galén publishing house, 2003. 248 pp. ISBN 80-7262-207-2.
- ↑ a b c d e MCLAUGHLIN, M R. Newborn Skin: Part II. Birthmarks [online]. American Family Physician, ©2008. [cit. 2012-09-04]. <https://www.aafp.org/afp/2008/0101/p56.html>.
- ↑ a b SALAVEC, M – ČELAKOVSKÁ, J – ŠIMKOVÁ, M. DYSPLASTIC NEVUS, DYSPLASTIC NEVUS SYNDROME AND OTHER MELANOCYTIC PIGMENT NEVUS. Dermatology for practice [online]. 2008, y. -, vol. -, p. 137-138, Available from <http://www.dermatologiepropraxi.cz/pdfs/der/2008/03/06.pdf>.